1 Drumond MC, Deen WM. Structural deter- minants of glomerular hydraulic permeabili- ty. Am J Physiol. 1994;266(1 Pt 2):F1–12.
2 Ikuma D, Hiromura K, Kajiyama H, Suwa J, Ikeuchi H, Sakairi T, et al. The correlation of urinary podocytes and podocalyxin with his- tological features of lupus nephritis. Lupus. 2018;27(3):484–93.
3 Faul C, Asanuma K, Yanagida-Asanuma E, Kim K, Mundel P. Actin up: regulation of podocyte structure and function by compo- nents of the actin cytoskeleton. Trends Cell Biol. 2007;17(9):428–37.
4 Yamaguchi Y, Iwano M, Suzuki D, Nakatani K, Kimura K, Harada K, et al. Epithelial-mes- enchymal transition as a potential explana- tion for podocyte depletion in diabetic ne- phropathy. Am J Kidney Dis. 2009;54(4):653– 64.
5 Miyauchi M, Toyoda M, Kobayashi K, Abe M, Kobayashi T, Kato M, et al. Hypertrophy and loss of podocytes in diabetic nephropathy. In- tern Med. 2009;48(18):1615–20.
6 Reidy K, Susztak K. Epithelial-mesenchymal transition and podocyte loss in diabetic kid- ney disease. Am J Kidney Dis. 2009;54(4): 590–3.
7 Sun D, Zhao X, Meng L. Relationship between urinary podocytes and kidney diseases. Ren Fail. 2012;34(3):403–7.
8 Al Hussain T, Al Mana H, Hussein MH, Akhtar M. Podocyte and parietal epithelial cell interactions in health and disease. Adv Anat Pathol. 2017;24(1):24–34.
9 Shankland SJ, Anders HJ, Romagnani P. Glo- merular parietal epithelial cells in kidney physiology, pathology, and repair. Curr Opin Nephrol Hypertens. 2013;22(3):302–9.
10 Hara M, Yanagihara T, Takada T, Itoh M, Matsuno M, Yamamoto T, et al. Urinary ex- cretion of podocytes reflects disease activity in children with glomerulonephritis. Am J Nephrol. 1998;18(1):35–41.
11 Vogelmann SU, Nelson WJ, Myers BD, Lem- ley KV. Urinary excretion of viable podocytes in health and renal disease. Am J Physiol Re- nal Physiol. 2003;285(1):F40–8.
12 Beyer-Boon ME, Voorn-den Hollander MJ. Cell yield obtained with various cytoprepara- tory techniques for urinary cytology. Acta Cy- tol. 1978;22(6):589–93.
13 Ohsaki H, Matsunaga T, Fujita T, Tokuhara Y, Kamoshida S, Sofue T. Quantifying podo- cytes and parietal epithelial cells in human urine using liquid-based cytology and WT1 Immunoenzyme staining. Bio Protoc. 2018; 8(9):e2827.
14 Fujita T, Sofue T, Moritoki M, Nishijima Y, Tokuhara Y, Wakisaka H, et al. Urinary WT1- positive cells as a non-invasive biomarker of crescent formation. Cytopathology. 2017; 28(6):524–30.
15 Gong Y, Symmans WF, Krishnamurthy S, Pa- tel S, Sneige N. Optimal fixation conditions for immunocytochemical analysis of estrogen receptor in cytologic specimens of breast car- cinoma. Cancer. 2004;102(1):34–40.
16 Davies RC, Calvio C, Bratt E, Larsson SH, La- mond AI, Hastie ND. WT1 interacts with the splicing factor U2AF65 in an isoform-depen- dent manner and can be incorporated into spliceosomes. Genes Dev. 1998;12(20):3217– 25.
17 Caricasole A, Duarte A, Larsson SH, Hastie ND, Little M, Holmes G, et al. RNA binding by the Wilms tumor suppressor zinc finger proteins. Proc Natl Acad Sci U S A. 1996; 93(15):7562–6.
18 Niksic M, Slight J, Sanford JR, Caceres JF, Hastie ND. The Wilms' tumour protein (WT1) shuttles between nucleus and cyto- plasm and is present in functional polysomes. Hum Mol Genet. 2004;13(4):463–71.
19 Hara M, Oohara K, Dai DF, Liapis H. Mitotic catastrophe causes podocyte loss in the urine of human diabetics. Am J Pathol. 2019;189(2): 248–57.
20 Lasagni L, Lazzeri E, Shankland SJ, Anders HJ, Romagnani P. Podocyte mitosis: a catas- trophe. Curr Mol Med. 2013;13(1):13–23.
21 Barisoni L, Mokrzycki M, Sablay L, Nagata M, Yamase H, Mundel P. Podocyte cell cycle regu- lation and proliferation in collapsing glomeru- lopathies. Kidney Int. 2000;58(1):137–43.
22 Hoshi S, Shu Y, Yoshida F, Inagaki T, Sonoda J, Watanabe T, et al. Podocyte injury pro- motes progressive nephropathy in zucker dia- betic fatty rats. Lab Invest. 2002;82(1):25–35.
23 Achenbach J, Mengel M, Tossidou I, Peters I, Park JK, Haubitz M, et al. Parietal epithelia cells in the urine as a marker of disease activ- ity in glomerular diseases. Nephrol Dial Transplant. 2008;23(10):3138–45.
24 Ohse T, Vaughan MR, Kopp JB, Krofft RD, Marshall CB, Chang AM, et al. De novo ex- pression of podocyte proteins in parietal epi- thelial cells during experimental glomerular disease. Am J Physiol Ren Physiol. 2010; 298(3):F702–11.
25 Wang P, Li M, Liu Q, Chen B, Ji Z. Detection of urinary podocytes and nephrin as markers for children with glomerular diseases. Exp Biol Med. 2015;240(2):169–74.
26 Wright RG, Halford JA. Evaluation of thin- layer methods in urine cytology. Cytopathol- ogy. 2001;12(5):306–13.
27 Kimura M, Toyoda M, Saito N, Kaneyama N, Miyatake H, Tanaka E, et al. A liquid-based cytology system, without the use of cytocen- trifugation, for detection of podocytes in urine samples of patients with diabetic ne- phropathy. J Diabetes Res. 2019; 2019: 9475637.
28 Kimura M, Toyoda M, Saito N, Kaneyama N, Miyatake H, Tanaka E, et al. Corrigendum to “A liquid-based cytology system, without the use of cytocentrifugation, for detection of podocytes in urine samples of patients with diabetic nephropathy”. J Diabetes Res. 2020; 2020:3623147.
29 Nakamura T, Ushiyama C, Suzuki S, Hara M, Shimada N, Ebihara I, et al. The urinary podo- cyte as a marker for the differential diagnosis of idiopathic focal glomerulosclerosis and minimal-change nephrotic syndrome. Am J Nephrol. 2000;20(3):175–9.
30 Hara M, Yanagihara T, Kihara I. Urinary podocytes in primary focal segmental glomer- ulosclerosis. Nephron. 2001;89(3):342–7.
31 Hara M, Yanagihara T, Kihara I. Cumulative excretion of urinary podocytes reflects disease progression in IgA nephropathy and Schön- lein-Henoch purpura nephritis. Clin J Am Soc Nephrol. 2007;2(2):231–8.