Amarasinghe AAPBN, Muhandiram MRS, Kodithuwakku SP, Thilakumara IP, Jayatilake
JAMS (2021). Identification, genotyping and invasive enzyme production of oral
Candida species from denture induced stomatitis patients and healthy careers. J
Oral Maxillofac Surgery, Med Pathol 33: 467–474.
Aoki S, Ito-Kuwa S, Nakamura Y, Masuhara T (1990). Comparative pathogenicity of a
wild-type strain and respiratory mutants of Candida albicans in Mice. Zentralblatt
fur Bakteriol 273: 332–343.
Dongari-Bagtzoglou A, Kashleva H (2003). Candida albicans triggers interleukin-8
secretion by oral epithelial cells. Microb Pathog 34: 169–177.
Fidel PL, Vazquez JA, Sobel JD (1999). Candida glabrata: Review of epidemiology,
pathogenesis, and clinical disease with comparison to C. albicans. Clin Microbiol
Rev 12: 80–96.
Hasebe A, Saeki A, Yoshida Y, Shibata K (2018). Differences in interleukin-1β
releaseinducing activity of Candida albicans toward dendritic cells and macrophages.
Arch Oral Biol 93: 115–125.
Hato H, Sakata K, Sato J, Hasebe A, Yamazaki Y, Kitagawa Y (2022). Factor associated
with oral candidiasis caused by co-infection of Candida albicans and Candida
glabrata: A retrospective study. J Dent Sci 17: 1458–1461.
Ikarashi K, Mano Y, Furuya N (2020). Comparative study of extracellular secretory
enzymes in Candida albicans and non-albicans Candida species. Japanese J Med
Technol 69: 69–74.
Jayampath Seneviratne C, Moe Thein Z, Duminda Jayasinghe R, et al (2015). Clinical
Appearance of oral Candida infection and therapeutic strategies. Front Microbiol
6: 1391.
Lewis MAO, Williams DW (2017). Diagnosis and management of oral candidosis. Br
Dent J 9: 675–681.
Luo G, Samaranayake LP, Yau JYY (2001). Candida species exhibit differential in vitro
hemolytic activities. J Clin Microbiol 39: 2971–2974.
Mane A, Gaikwad S, Bembalkar S, Risbud A (2012). Increased expression of virulence
attributes in oral Candida albicans isolates from human immunodeficiency
viruspositive individuals. J Med Microbiol 61: 285–290.
Manns JM, Mosser DM, Buckley HR (1994). Production of a hemolytic factor by
Candida albicans. Infect Immun 62: 5154–5156.
Oouchi M, Hasebe A, Hata H, et al (2015). Age-related alteration of expression and
function of TLRs and NK activity in oral candidiasis. Oral Dis 21: 645–651.
Price MF, Wilkinson ID, Gentry LO (1982). Plate method for detection of phospholipase
activity in Candida albicans. Sabouraudia 20: 7–14.
Sardi JCO, Duque C, Fling Ö, Gon RB, Alves Ç (2012). Genetic and phenotypic
evaluation of Candida albicans strains isolated from subgingival biofi lm of
diabetic patients with chronic periodontitis. Med Mycol 50: 467–475.
Shekhany K, Jalal P (2022). Identification and genotyping of Candida species involved
in oral candidiasis among diabetic patients. Sulaimani Dent J 9: 45–53.
Silvania, Henriques M, Hayes A, Oliveira R, Azeredo J, Williams DW (2011). Candida
glabrata and Candida albicans co-infection of an in vitro oral epithelium. J Oral v
Soysa NS, Samaranayake LP, Ellepola ANB (2006). Diabetes mellitus as a contributory
factor in oral candidosis. Diabet Med 23: 455–459.
Steele C, Fidel PL (2002). Cytokine and chemokine production by human oral and
vaginal epithelial cells in response to Candida albicans. Infect Immun 70: 577–
583.
Suryana K, Suharsono H, Gede I, Antara PJ (2020). Factors associated with oral
candidiasis in people living with HIV/AIDS: A Case Control Study. HIV/AIDS 14:
33–39.
Talapko J, Juzbaši´c MJ, Matijevi´cmatijevi´c T, et al (2021). Candida albicans-The
virulence factors and clinical manifestations of infection. J Fungi 7: 79.
Tamura M, Watanabe K, Mikami Y, Yazawa K, Nishimura K (2001). Molecular
characterization of new clinical isolates of Candida albicans and C. dubliniensis in
Japan: analysis reveals a new genotype of C. albicans with group I intron. J Clin
Microbiol 39: 4309–4315.
Villar CC, Kashleva H, Dongari-Bagtzoglou A (2004). Role of Candida albicans
polymorphism in interactions with oral epithelial cells. Oral Microbiol Immunol
19: 262–269.
Villar CC, Zhao XR (2010). Candida albicans induces early apoptosis followed by
secondary necrosis in oral epithelial cells. Mol Oral Microbiol 25: 215–225.
Zdanavičienė E, Sakalauskienė J, Gleiznys A, Gleiznys D, Žilinskas J (2017). Host
responses to Candida albicans. A review. Balt Dent Maxillofac J 19: 109–132.
Ziccardi M, Souza LOP, Gandra RM, et al (2015). Candida parapsilosis (sensu lato)
isolated from hospitals located in the Southeast of Brazil: Species distribution,
antifungal susceptibility and virulence attributes. Int J Med Microbiol 305: 848–
859.
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