「Indoxyl Sulfate-induced Vascular Calcification is mediated through Altered Notch Signaling Pathway in Vascular Smooth Muscle Cells」の関連論文
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子宮内膜癌におけるPPP2R1A遺伝子変異の意義の検討
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C-type lectin Mincle mediates cell death–triggered inflammation in acute kidney injury
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オリゴデンドロサイト前駆細胞の制御機構および病態時の機能に関する研究
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TIMP-1は消化管線維化における新たな指標となる
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Direct reprogramming of adult adipose-derived regenerative cells toward cardiomyocytes using six transcriptional factors
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Primate-specific POTE-actin gene could play a role in human folliculogenesis by controlling the proliferation of granulosa cells
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慢性呼吸器疾患におけるIL-17F及びNrf2の機能解析
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アポトーシス進行中のBlebにおける形質膜と細胞質の協調的な変化
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Zonisamide ameliorates neuropathic pain partly by suppressing microglial activation in the spinal cord in a mouse model
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X連鎖性好中球減少症(XLN)の病態に関する研究
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Wnt非古典経路におけるシグナル伝達分子機構の解析
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マクロファージとの相互作用により誘導されるYKL-40/osteopontin-integrin β4-p70S6K経路の早期食道扁平上皮癌における生物学的・臨床的な意義
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Downregulation of ROBO4 in Pancreatic Cancer Serves as a Biomarker of Poor Prognosis and Indicates Increased Cell Motility and Proliferation Through Activation of MMP-9
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細胞外小胞によるがん悪性化機構の解明に関する研究 (本文)
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CD163陽性M2マクロファージはTLR7/IRAK4/NFkBシグナルを介してIgG4関連疾患の線維化を促進する
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Zonisamide upregulates neuregulin-1 expression and enhances acetylcholine receptor clustering at the in vitro neuromuscular junction
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T細胞リンパ腫におけるマイクロRNAによるがんとがん微小環境の制御
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非小細胞肺がん患者におけるオシメルチニブ有害事象とSTAT3, CYP3A5, ABCG2遺伝子多型との関連
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膵β細胞におけるグルコース応答性転写因子ChREBPの転写制御因子の探索および機能解析
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パーキンソン病の新規リスク遺伝子Midnolinの発現調節機構と病態生理的な役割の解明
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多発性嚢胞腎モデルラットの肝病変に対する長期的運動とメトホルミンの効果
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Expression of Vascular Endothelial Growth Factor by Retinal Pigment Epithelial Cells Induced by Amyloid-β Is Depressed by an Endoplasmic Reticulum Stress Inhibitor
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Serum exosomal miR-638 is a prognostic marker of HCC via downregulation of VE-cadherin and ZO-1 of endothelial cells
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Apelin/APJ signaling suppresses the pressure ulcer formation in cutaneous ischemia-reperfusion injury mouse model
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A novel renal perivascular mesenchymal cell subset gives rise to fibroblasts distinct from classic myofibroblasts
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Downregulated ATP6V1B1 expression acidifies the intracellular environment of cancer cells leading to resistance to antibody-dependent cellular cytotoxicity
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Successful treatment of a novel type I interferonopathy due to a de novo PSMB9 gene mutation with a Janus kinase inhibitor
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Photosensitizer verteporfin inhibits the growth of YAP‑ and TAZ‑dominant gastric cancer cells by suppressing the anti‑apoptotic protein Survivin in a light‑independent manner
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BRCA1 転写共役因子MED1の DNA損傷修復機構における機能解析
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CD44高発現口腔扁平上皮癌細胞におけるamoeboid-to-mesenchymal transitionの制御機構
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CD44高発現口腔扁平上皮癌細胞におけるamoeboid-to-mesenchymal transitionの制御機構
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Hepatic stellate cells in hepatocellular carcinoma promote tumor growth via growth differentiation factor 15 production
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CTGF/CCN2 facilitates LRP4-mediated formation of the embryonic neuromuscular junction
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GPR120を介した新規アミロイドβ分解制御機構の解明
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miR-27b inhibits hepatic differentiation via antagonizing BMP signaling in early differentiation of human induced pluripotent stem cells.
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歯肉幹細胞由来エクソソームは、M2分化を誘導し、歯槽骨吸収を抑制した
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Microphthalmia-Associated Transcription Factor-Dependent Melanoma Cell Adhesion Molecule Activation Promotes Peritoneal Metastasis of Ovarian Cancer
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CARD9の肺腺がんの新しい予後因子としての可能性
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The role of CUB domain-containing protein 1 and MET interaction in invasion of breast cancer cells
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酸化ストレスによるマクロファージ機能変化が歯周病の進行に与える影響
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Oligodendrogenesis and myelin formation in the forebrain require platelet-derived growth factor receptor-alpha
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Staurosporine and venetoclax induce the caspase-dependent proteolysis of MEF2D-fusion proteins and apoptosis in MEF2D-fusion (+) ALL cells
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膵癌の薬剤抵抗性におけるKEAP1-Nrf2経路の意義に関する検討
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Blockade of EGFR improves responsiveness to PD-1 blockade in EGFR-mutated non-small cell lung cancer
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好中球Protein Kinase R(PKR)を介した腎臓炎症惹起メカニズムの解明
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Barrett食道癌周囲のBarrett粘膜におけるGlucocorticoid receptor (GR) 発現と粘膜固有層リンパ球浸潤に関する検討
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Increased numbers but reduced resorption activity of osteoclasts in toll-like receptor 2 (TLR2) deficient mice [an abstract of entire text]
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miR-1260bは小胞体ストレス制御関連のATF6βを介して歯周炎の骨吸収を抑制する
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High-fat Feeding Causes Inflammation and Insulin Resistance in the Ventral Tegmental Area in Mice
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マルファン症候群の大動脈瘤形成における性差の分子機構